The popular story of how low-carb diets work goes something like this: Reducing your carbohydrate...
Let me introduce myself. My name is Mark Sisson. I’m 63 years young. I live and work in Malibu, California. In a past life I was a professional marathoner and triathlete. Now my life goal is to help 100 million people get healthy. I started this blog in 2006 to empower people to take full responsibility for their own health and enjoyment of life by investigating, discussing, and critically rethinking everything we’ve assumed to be true about health and wellness...Tell Me More
For today’s edition of Dear Mark, I’m answering three questions from readers. First up, what are we to make of a recent study claiming to show that “high-fat diets” are harmful to rodents? Is it truly a high-fat diet, and what does it mean for us? For the second question, I field a comment from a reader experiencing a confluence of troubling symptoms and test results on his keto diet. And third, does keto actually cure diabetes, or just manage it?
If you think of Type 2 diabetes as carbohydrate intolerance, the natural dietary response should be to restrict the offending dietary component. And when this occurs—when diabetic patients restrict carbs—their symptoms improve, often to a greater degree than diabetic patients on other diets. Keto restricts more carbs than even other low-carb diets, so on the face of things, keto seems great for diabetes.
Let’s take a closer look.
Almost everyone has at least one dietary restriction. Maybe your religion or cultural traditions prohibit specific foods or food pairings. Maybe your physiological response to certain foods—an allergy or intolerance—prevent you from eating them. Or perhaps your immediate goals preclude a food’s inclusion in your diet.
Like every other diet, keto is already circumscribed by basic principles, which can make further limitations difficult to accommodate. But the benefits of going keto, at least for part of the time, are well-established and worth the effort. You want to do it. How can you go keto while honoring your own dietary bounds?
It depends on the restriction.
For today’s edition of Dear Mark, I’m answering three questions from readers. All three questions come from last week’s saturated fat post. First, I explore the true reason for increases in serum palmitic acid—too many carbs. Second, I investigate whether dairy or saturated fat affect polyphenol absorption, and whether it actually matters. And finally, I discuss the merits of avocado oil for high heat cooking and searing.
No matter what kind of evidence comes out to the contrary, the anti-saturated fat sect won’t relinquish its dogma. Whenever its advance is rebuffed—perhaps by an observational study showing the lack of relations between saturated fat intake and heart disease, or a study showing the beneficial effects of saturated fat on multiple health markers—they regroup and try another route. The latest is a study that several readers sent to me, worried that the attack had finally made it through the defenses. In it, researchers purport to show that saturated fat increases the solidity and rigidity of cellular membranes, reducing membrane fluidity and eventually leading to cell death.
Is it true? Have we finally lost? What was this study all about?
The explosive growth of interest in the ketogenic diet has been a net good for the state of nutrition. For one, people have accepted the fact that eating fat won’t kill you, and they’re even getting clued into the benefits of eating it.
But there are places where I part ways with the popular keto movement.
Let me explain….