It’s impossible to talk about using food as a drug without looking at the genuine neurological and hormonal impacts it has on the body. The fact is, certain foods affect us more like drugs than others.
With actual drug use, we’re not operating with innate satiation signaling. But with food, our bodies have a built-in system for telling us when to eat, how much to eat and when to stop.
In our paleolithic ancestors’ time, it worked great. Today, we’ve become our own saboteurs. We’ve known for years that sugary and processed foods (those that strategically combine sugar, salt and certain fats into a triple crown disaster) are intentionally designed to override our inherent satiation signals and hyper-trip our reward systems.
Keto may not be for everyone, and low-carb is not the only way to eat well, but most would agree that people in the modern world tend to eat way too many carbohydrates—far more than their lifestyles and activity levels warrant. Along with some other big factors, excessive intake of refined carbohydrates is a major player in the modern epidemics of obesity, diabetes, heart disease, and other disorders. This is no longer controversial. Reducing carbs is a good move for most folks.
The majority of my readers are on some kind of low carb diet. Maybe they’re not fully keto. But they all tend to acknowledge the utility of limiting one’s carbs to only those they need. One of the more common questions I receive from this group concerns carb cycling—periodically adding more carbs to an otherwise low-carb diet.
Most discussion of chronically-elevated insulin levels (hyperinsulinemia) and insulin resistance revolves around their relationship to bodyweight. This is unsurprising. Bodyweight’s what “sells tickets.” It’s why most people get interested in diet, health, fitness, and nutrition—to lose weight or avoid gaining it.
But improving insulin sensitivity and reducing fasting insulin levels have major ramifications for your health, longevity, and resistance to disease. And it’s not just because “weight gain is unhealthy.” Insulin itself, in excess, exerts seriously damaging effects. Today, I want to impress upon you the importance of controlling your insulin response by laying out some of the health problems that stem from not controlling it.
For today’s edition of Dear Mark, I’m answering some questions about keto (hey, you folks keep asking!). First, is being on a ketogenic diet actually congruent with our ancestry? Is there historical precedent? Next, is bad breath really a reliable indication of being in ketosis? And finally, could going keto help treat the autoimmune disease lupus?
Let’s take a look:
The explosive growth of interest in the ketogenic diet has been a net good for the state of nutrition. For one, people have accepted the fact that eating fat won’t kill you, and they’re even getting clued into the benefits of eating it.
But there are places where I part ways with the popular keto movement.
Let me explain….
Good morning, folks. With next week’s The Keto Reset Diet release, I’ve got keto on the mind today—unsurprisingly. I’ve had a lot of questions lately on duration. As I’ve mentioned before, a good six weeks of ketosis puts in place all the metabolic machinery for lasting adaptation (those extra mitochondria don’t evaporate if/when you return to traditional Primal eating).
But what about the other end of the issue? How long is too long? I don’t do this often, but today I’m reposting an article from a couple of years ago on this very topic. I’ve added a few thoughts based on my recent experience. See what you think, and be sure to share any lingering questions on the question of keto timing and process. I’ll be happy to answer them in upcoming posts and Dear Mark columns.