In Monday’s “Dear Mark” post, I briefly outlined a few of the benefits to having healthy, abundant mitochondria, and in the past, I’ve alluded to the damaging effects of statins on mitochondrial function. All good, yeah, but a couple brief paragraphs in the middle of a Monday post aren’t enough. Mitochondrial function and mitochondrial biogenesis – the growth of new mitochondria – deserve more than that. Like, their own post. Today, I’m going to dig a little deeper. I’m going to lay out why growing more and healthier mitochondria (mitochondrial biogenesis) is good for your health, your longevity (and compression of morbidity), and your energy levels. I’ll explain why becoming a fat-burning beast optimizes mitochondrial function, and I’ll go over why this is so important if you’re looking to transform your body.
You know how we say that grains exist on a spectrum of suitability, from “really bad” wheat to “not so terrible” rice? Well, what about the rest of ‘em? They may be the most commonly consumed (and thus encountered) grains, but wheat and rice aren’t the only grains on the spectrum. Since I get a lot of email about oats, I figured they were a good choice for this post. Besides – though I was (and still mostly am) content to toss the lot of them on the “do not eat” pile, I think we’re better served by more nuanced positions regarding grains. Hence, my rice post. Hence, my post on traditionally prepared grains. And hence, today’s post on oats. Not everyone can avoid all grains at all times, and not everyone wants to avoid all grains at all times. For those situations, it makes sense to have a game plan, a way to “rank” foods.
Today, we’ll go over the various incarnations of the oat, along with any potential nutritional upsides or downsides. But first, what is an oat?
We interrupt our regularly scheduled Friday Success Story to bring you a timely and critical look at this week’s Hottest Health Headline. And who better to tackle the research in question than expert study-dismantler Denise Minger? You may remember Denise from the recent article she wrote for MDA in which she went toe-to-toe with a study linking a high fat diet with breast cancer. Today she takes on our nemesis, our foe, our mortal enemy – the Whole Grain. And now, Denise…
A headline-grabbing study just hit the press, and on the surface, it looks like a home run for team Healthy Whole Grain. This chunk of research – officially titled “Dietary Fiber Intake and Mortality in the NIH-AARP Diet and Health Study” – followed a pool of over half a million adults and found that, across the board, the folks eating the most fiber had lower rates of death from almost every disease. But here’s the kicker: The only fiber that seemed to boost health was the kind from grains. Not veggie fiber. Not fruit fiber. Just grains, grains, grains.
I’ve been on a bit of an alternative sweetener kick these past few weeks, for good reason: people want and need to know about this stuff. While a purist shudders at the prospect of any non- or hypo-caloric sugar substitute gracing his or her tongue, I’m a realist. People are going to partake and it’s important to understand what’s entering your body and what, if any, effects it will have. Whether it’s diet soda, artificial sweeteners, stevia, or the mysterious sugar alcohols, people want the sweet without worrying about a big physiological effect – an insulin surge, a blood glucose dip, even a migraine. So I’ve been covering the various types and have tried to be comprehensive about it. As a whole, it all seems fairly safe. Alternative sweeteners might mess with some folks’ adherence to a low-sugar diet, and they might induce or fortify cravings, but the research doesn’t suggest that they’re going to give you cancer or diabetes. The potentially negative effects are all fairly subjective, so it’s safe to play around with them and determine their role in your life based on how they affect your appetite, state-of-mind, and any other subjective health markers.
The notion that artificial sweeteners (and sweet tastes in general) might produce an insulin response is one of those murky memes that winds itself around the blogs, but it’s never stated one way or the other with any sort of confidence. I briefly mentioned the possibility of non-caloric sweeteners influencing satiety hormones in last week’s diet soda post, and a number of you guys mentioned the same thing. Still, I’ve never seen unequivocal evidence that this is the case.
This whole idea first came to my attention some time ago when my dog Buddha got into a bottle of “alternative sleep assists” which contained, among other things, 5 HTP (version of l-tryptophan) and xylitol (sugar alcohol). Long story short, dogs can’t take xylitol because it causes a spike in insulin, which then severely depletes blood glucose. Buddha got past this with a trip to the vet’s at 10:30 Sunday night (thanks, Dr. Dean). But it occurred to me that the same effect might be seen in humans, which is why I pose the question today…
Do artificial sweeteners induce insulin secretion (perhaps via cephalic phase insulin release, which is sort of the body’s preemptive strike against foods that will require insulin to deal with)?
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